Neuropsychological performance of patients in treatment for cocaine addiction / Rendimiento neuropsicológico de pacientes en tratamiento por adicción a la cocaína
DOI:
https://doi.org/10.21134/haaj.v8i1.65Keywords:
Adicción a la cocaína, Déficit neuropsicológico, Funciones ejecutivas. / Keywords, Cocaine addiction, Neuropsychological deficit, Executive functions.Abstract
En este estudio se evaluó la presencia de alteraciones neuropsicológicas en 50 pacientes adictos a la cocaína en comparación con un grupo control de 22 sujetos de la población normal, balanceados en edad, sexo y años de escolarización. Se aplicó un protocolo de evaluación compuesto de tareas sensibles a la detección de alteraciones en diferentes componentes de las funciones ejecutivas. Los resultados descartaron la presencia de un déficit neuropsicológico generalizado en la población de adictos a la cocaína, y apuntaron hacia alteraciones neuropsicológicas más específicas.
En concreto, el grupo clínico obtuvo un peor rendimiento en tareas relacionadas con el procesamiento de la información e inhibición de respuestas, así como una tendencia hacia una menor flexibilidad mental. Además, el consumo abusivo concomitante de alcohol interfería en funciones
de procesamiento y recuperación de la información.
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Abstract
The aim of this study was to assess the presence of neuropsychological alterations in 50 patients addicted to cocaine, compared to a control group of 22 people from the normal population, balanced in age, sex and years of education. We applied an assessment protocol comprising tasks sensitive to the detection of alterations in different components of the executive functions. The results ruled out the presence of a generalized
neuropsychological deficit in the population of cocaine addicts, and indicated more specific neuropsychological alterations. In particular, the clinical group performed more poorly in tasks related to information processing and to response inhibition, and showed a tendency for less mental flexibility. Moreover, concomitant alcohol abuse interfered with functions of information processing and recovery.
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